In my opinion, based on research, Covid is not a "virus" - it is a misdiagnosed disease, or a set of symptoms caused NOT by a virus, but by toxicity of used substance/s, especially graphene. For reference:
In my opinion, based on research, Covid is not a "virus" - it is a misdiagnosed disease, or a set of symptoms caused NOT by a virus, but by toxicity of used substance/s, especially graphene. For reference:
Oxidative stress being the real cause of the so-called Covid complications and deaths is killing or damaging these people - especially those most at risk (adding to the mistreatment as the disease is misdiagnosed and thus treated with toxic drugs such as Remdesivir, rather than addressing oxidative stress with antioxidants).
The occurrence of this oxidative stress is one of the main mechanisms of graphene/nanotechnology toxicity.
https://particleandfibretoxicology.biomedcentral.com/articles/10.1186/s12989-016-0168-y “ We also point out that various factors determine the toxicity of GFNs including the lateral size, surface structure, functionalization, charge, impurities, aggregations, and corona effect ect. In addition, several typical mechanisms underlying GFN toxicity have been revealed, for instance, physical destruction, oxidative stress, DNA damage, inflammatory response, apoptosis, autophagy, and necrosis. In these mechanisms, (toll-like receptors-) TLR-, transforming growth factor β- (TGF-β-) and tumor necrosis factor-alpha (TNF-α) dependent-pathways are involved in the signalling pathway network, and oxidative stress plays a crucial role in these pathways. In this review, we summarize the available information on regulating factors and the mechanisms of GFNs toxicity, and propose some challenges and suggestions for further investigations of GFNs, with the aim of completing the toxicology mechanisms, and providing suggestions to improve the biological safety of GFNs and facilitate their wide application.”
“ROS production leading to oxidative stress
Oxidative stress had a significant role in GO-induced acute lung injury [30], and the inflammatory responses caused by oxidative stress often emerged upon exposure to GFNs
The treatment that should follow because of the toxicity of the substances used - (nanotechnology is used in masks, flu "vaccines", PCR "tests", chemtrails, foods, different drugs and of course and most importantly in C-19 injections), are antioxidants. There should be prevention and treatment of oxidative stress. Oxidative stress is the reason why people have low saturation, and as a result of severe oxidative stress, cells are destroyed, apoptosis occurs, that is, cell death and a further cascade of events:
clotting, organ failure, heart attacks, strokes, etc.
The virus (or just a regular flu that triggers these symptoms) seems to be pretty secondary to graphene toxicity. This is nanotechnology that crosses all blood barriers, is toxic, causes oxidative stress and injures/kills people.
So, according to these studies on graphene toxicity, Covid is not a virus, but yes, people, poisoned by nanotechnology, because of free and oxy radicals present, are susceptible to infections or flu, and these infections, as additional oxidative stress - and strain on their already struggling organisms - can be fatal to them.
When Pfizer was asked how/where and for how long "Spike Protein" is produced, it admitted they had no idea. Spike Protein seems to be just a cover for injecting people and creating a fear of pandemics. This doesn't mean that a protein or even Spike Protein can't also be used in these vaccines, but if it is used, its functions seem to have nothing to do with creating antibodies to the "virus". But it may cause formation of misfolded proteins, AMYLOIDOSIS.
Thus, Covid is a misdiagnosed disease. It seems to be first and foremost graphene toxicity poisoning with all that come from this toxicity such as physical destruction, oxidative stress, DNA damage, inflammatory response, apoptosis, autophagy, and necrosis. Those are causing blood clots, organ failure, strokes, and many other injuries, including death. Graphene can result in acute inflammation response and chronic injury by interfering with the normal physiological functions of important organs. Studies regarding risks of graphene in the brain show that graphene application leads to harmful effects on brain tissue development and the atypical ultrastructure was observed in the brain. Graphene demonstrates its toxicity in the central nervous system and toxicity in reproduction and development system. In the animal studies the pregnant mice had abortions at all doses, and most pregnant mice died when the high dose was injected during late gestation and the development of offspring was delayed during the lactation period. The high dose reduces milk production and postpones the growth of offspring. The developmental toxicity of graphene induces structural abnormalities, growth retardation, behavioral and functional abnormalities, and even death. Graphene induces the lung injury with inflammatory cell infiltration, pulmonary edema and granuloma formation in the lungs. Graphene causes cytotoxic effects and mitochondrial injury, leads to inflammations, induces DNA damage, decreases cell adhesion and induces cell apoptosis - cell death. Graphene inserts between the base pairs of double-stranded DNA and disturb the flow of genetic information at the molecular level, which is the main cause of its mutagenic effect. It is hemotoxic, cytotoxic, cardiotoxic, neurotoxic, harmful to reproductive system. Graphene sharpened edges cause physical destruction...
There are other toxic ingredients and heavy metals in those vials:
There is also toxic PEG. All of them will result in oxidative stress or acute oxidative stress.
https://pubmed.ncbi.nlm.nih.gov/30641367/ How graphene affects the misfolding of human prion protein: A combined experimental and molecular dynamics simulation study "Our simulations suggest that prion protein can be quickly and tightly adsorbed onto graphene together with the weak conformational rearrangement and may reorient when approaching the surface. The Van der Waals' force drive the adsorption process. In the induction of graphene, H1 and S2-H2 loop regions of prion become unstable and prion begins to misfold partially. Our work shows that graphene can induce the misfolding of prion protein and may cause the potential risk to biosystems."
So, for treatment - Glutathione precursors seem to be the most important because Glutathione is the master of all other antioxidants.
NAC helps dissolve amyloid plaque formations and blood clots and is one of the Glutathione precursors.
To address both oxidative stress and amyloid plaque formation, this is what would be helpful: Antioxidants!!! NAC, Zinc - and its ionophores, such, as Ivermectin, if available, Quercetin, Vitamin C-and others: Q10, K2MK7, Vitamin E, Resveratrol, Vitamin D, Beta Carotene, Nigella Sativa, Turmeric, etc.
The study by CHOP researchers suggests NAC may block the precipitation of amyloid plaque deposits, as well as help break up their formation
“Amyloids cannot precipitate without aggregating, so if we can prevent that aggregation with a drug [NAC] that is already available, then we could make an incredible difference in the lives of these patients.
Additionally, since we already have genetic testing available to identify these patients, we could conceivably give this treatment early in life and potentially prevent that first stroke from ever occurring.”
In the Pfizer Document - Postmarketing Experience, different Amyloidosis as an adverse result of vaccination is mentioned as well - including Cardiac Amyloidosis and Renal, Skin Amyloidosis - etc.
Here is the information what markers can be done to find out if oxidative stress is present: A limitation of our study, apart from the small sample size however, is that we were unable to do laboratory testing in our patients, including checking oxidative stress markers (lipid peroxides) as well as inflammatory markers (CRP, ferritin, D-dimer) and LDH which might demonstrate a change post GSH administration
In addition, several typical mechanisms underlying GFN toxicity have been revealed, for
instance, physical destruction, oxidative stress, DNA damage, inflammatory response,
apoptosis, autophagy, and necrosis. In these mechanisms, (toll-like receptors-) TLR-,
transforming growth factor β- (TGF-β-) and tumor necrosis factor-alpha (TNF-α) dependent pathways are involved in the signaling pathway network, and oxidative stress plays a
crucial role in these pathways.
Another study suggested that the irregular protrusions and sharp edges of the nanosheets could damage the plasma membrane, thus letting G entering the cell by piercing the phospholipid-bilayer (Li Y. et al., 2013). These features raise additional safety concerns, as free GRMs in the cytoplasm may lead to disruption of the cytoskeleton, impaired cell motility and blockade of the cell-cycle, similar to carbon nanotube-induced cytotoxicity. Exposed larvae displayed GO in the CNS and, most importantly, an induction of Parkinson’s disease-like symptoms such as disturbance of locomotor activity, dopaminergic neurons loss and formation of Lewy bodies. These effects were likely a consequence of mitochondrial damage and apoptosis through the caspase 8 pathway, in the presence of a more general metabolic disturbance
Two recent studies give us a less than rosy angle. In the first, a team of biologists, engineers and material scientists at Brown University examined graphene’s potential toxicity in human cells. They found that the jagged edges of graphene nanoparticles, super sharp and super strong, easily pierced through cell membranes in human lung, skin and immune cells, suggesting the potential to do serious damage in humans and other animals.
Generally, GFNs may exert different degrees of toxicity in animals or cell models by following with different administration routes and penetrating through physiological barriers, subsequently being distributed in tissues or located in cells, eventually being excreted out of the bodies. This review collects studies on the toxic effects of GFNs in several organs and cell models. We also point out that various factors determine the toxicity of GFNs including the lateral size, surface structure, functionalization, charge, impurities, aggregations, and corona effect ect. In addition, several typical mechanisms underlying GFN toxicity have been revealed, for instance, physical destruction, oxidative stress, DNA damage, inflammatory response, apoptosis, autophagy, and necrosis.
If you consider the implications of the DOD developing the Pfizer jab as a prototype countermeasure as described by Sasha Latypova, the DOD can instruct Pfizer to vary the contents of the inoculations as investigation of prototypes. The implication is that some vials will contain graphene and some not. Dr. Ryan Cole in conjunction with a mass spec lab recent analyzed a number of vials and found no graphene in any of them and found no mRNA is some of them as well (essential vials of saline). Others contained degraded mRNA with varying percentages of intact "jab spike protein" mRNA.. My interpretation is the jabs are a combination of different DOD prototypes and poor manufacturing quality control On another note, I appreciate all your links to research on graphene. Graphene is clearly toxic and it reportedly used in the limpid nanoparticles that come from a Chinese manufacturer (they state this on their website) as noted by Karen Kingston. With regard to ROS, toxins and viruses and pathogenic bacteria also elevate ROS. That graphene could be synergistic with pathogens in elevating ROS and misfolding of proteins and creating and inducing amyloid proteins is not beyond a possibility. Dr. Arne Bukhardt's autopsy biopsy slides clearly show amyloid infiltration of critical organs including the heart and brain and well as the spike protein "mRNA infections" triggering killer lymphocyte attacks on infected cells and consequent scarring.
So, the presence of toxic graphene can cause amyloidosis. However, if the technology used in these injections is also based on peptides (as has been widely reported in the scientific literature: Self-assembling peptide semiconductors | https://www.science.org/doi/10.1126/science.aam9756), the production of amyloid in the bone marrow can lead to severe amyloidosis and related complications and death, including the currently manifested so-called sudden death syndrome. The key question, therefore, is also what technology was used in these injections.
“Short peptides, specifically those containing aromatic amino acids, can self-assemble into a wide variety of supramolecular structures that are kinetically or thermodynamically stable; the representative models are diphenylalanine and phenylalanine-tryptophan. Different assembly strategies can be used to generate specific functional organizations and nanostructural arrays, resulting in finely tunable morphologies with controllable semiconducting characteristics. Such strategies include molecular modification, microfluidics, coassembly, physical or chemical vapor deposition, and introduction of an external electromagnetic field (EMF).
Self-assembling peptide nanomaterials may serve as an alternative source for the semiconductor industry because they are eco-friendly, morphologically and functionally flexible, and easy to prepare, modify, and dope. Moreover, the diverse bottom-up methodologies of peptide self-assembly facilitate easy and cost-effective device fabrication, with the ability to integrate external functional moieties. For example, the coassembly of peptides and electron donors or acceptors can be used to construct n-p junctions, and vapor deposition technology can be applied to manufacture custom-designed electronics and chips on various substrates.
The inherent bioinspired nature of self-assembling peptide nanostructures allows them to bridge the gap between the semiconductor world and biological systems, thus making them useful for applications in fundamental biology and health care research. Short peptide self-assemblies may shed light on the roles of protein semiconductivity in physiology and pathology. For example, research into the relationship between the semiconductive properties of misfolded polypeptides characteristic of various neurodegenerative diseases and the resulting symptoms may offer opportunities to investigate the mechanisms controlling such ailments and to develop therapeutic solutions. Finally, self-assembling short peptide semiconductors could be used to develop autonomous biomachines operating within biological systems. This would allow, for example, direct, label-free, real-time monitoring of a variety of metabolic activities, and even interference with biological systems.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5457452/ “Peptide based nano-assemblies with their self-organizing ability has shown lot of promise due to their high degree of thermal and chemical stability, for biomaterial fabrication. Developing an effective way to control the organization of these structures is important for fabricating application-oriented materials at the molecular level. The present study reports the impact of electric and magnetic field-mediated perturbation of the self-assembly phenomenon, upon the chemical and structural properties of diphenylalanine assembly. Our studies show that, electric field effectively arrests aggregation and self-assembly formation, while the molecule is allowed to anneal in the presence of applied electric fields of varying magnitudes, both AC and DC. The electric field exposure also modulated the morphology of the self-assembled structures without affecting the overall chemical constitution of the material. Our results on the modulatory effect of the electric field are in good agreement with theoretical studies based on molecular dynamics reported earlier on amyloid forming molecular systems.”
“In last decade, there has been an increased focus on organic and bio-organic nano-assemblies. Peptide nanotubes, their physical properties, and assembly morphologies are extensively studied due to their excellent biocompatibility as well as functional and structural diversity. Many ordered supramolecular structures have been constructed using peptides as the building blocks. The most extensively utilized peptide-based building block is diphenylalanine (Phe-Phe or FF), which is the shortest bio-molecule known to self-assemble into ordered nanostructures. FF incidentally is also the core recognition motif of the β-amyloid polypeptide, a peptide associated with Alzheimer’s disease1. It can self-assemble into a variety of structures like microtubes, nanotubes2, microcrystals, nanofibers3, nanorods4, 5 and nanowires6.”
“The potential of these supramolecular structures have been utilized in diverse fields including nanofabrication, drug delivery vehicles7, bio-sensing8, energy storage devices, and hydrogels for tissue engineering”.
“One of the key challenges in the field of supramolecular chemistry has been controlling the self-assembly of molecules into ordered functional units. Previously, a number of strategies including pH mediated control21, 22, solvent mediated control23, covalent modifications24, vapour deposition25, 26, temperature27, surface28, 29, relative humidity30, symmetry31 and magnetite coating on the surface of nanotubes32 have been employed to regulate the architecture of diphenylalanine self-assemblies.”
Andrij Baumketner in a recent study, explored the feasibility of using external electric field to disaggregate amyloid fibrils, by inducing folding into an α-helical state reducing their β sheet conformation38. This is especially important because FF is the core recognition motif of β-amyloid segment. Here in this study, we attempt to confirm the effect of AC (Alternating Current) and DC (Direct Current) electric field on diphenylalanine self-assembly using experimental approach.
Different assembly strategies can be used to generate specific functional organizations and nano structural arrays, resulting in finely tunable morphologies with controllable semiconducting characteristics. Such strategies include molecular modification, microfluidics, subassembly, physical or chemical vapor deposition, and introduction of an external electromagnetic field.
Regarding Covid:
In my opinion, based on research, Covid is not a "virus" - it is a misdiagnosed disease, or a set of symptoms caused NOT by a virus, but by toxicity of used substance/s, especially graphene. For reference:
https://outraged.substack.com/p/can-toxic-substances-be-mandated
https://outraged.substack.com/p/graphenenanotechnology-in-masks-and
https://outraged.substack.com/p/problem-and-solution?utm_source=profile&utm_medium=reader2
Oxidative stress being the real cause of the so-called Covid complications and deaths is killing or damaging these people - especially those most at risk (adding to the mistreatment as the disease is misdiagnosed and thus treated with toxic drugs such as Remdesivir, rather than addressing oxidative stress with antioxidants).
The occurrence of this oxidative stress is one of the main mechanisms of graphene/nanotechnology toxicity.
https://particleandfibretoxicology.biomedcentral.com/articles/10.1186/s12989-016-0168-y “ We also point out that various factors determine the toxicity of GFNs including the lateral size, surface structure, functionalization, charge, impurities, aggregations, and corona effect ect. In addition, several typical mechanisms underlying GFN toxicity have been revealed, for instance, physical destruction, oxidative stress, DNA damage, inflammatory response, apoptosis, autophagy, and necrosis. In these mechanisms, (toll-like receptors-) TLR-, transforming growth factor β- (TGF-β-) and tumor necrosis factor-alpha (TNF-α) dependent-pathways are involved in the signalling pathway network, and oxidative stress plays a crucial role in these pathways. In this review, we summarize the available information on regulating factors and the mechanisms of GFNs toxicity, and propose some challenges and suggestions for further investigations of GFNs, with the aim of completing the toxicology mechanisms, and providing suggestions to improve the biological safety of GFNs and facilitate their wide application.”
“ROS production leading to oxidative stress
Oxidative stress had a significant role in GO-induced acute lung injury [30], and the inflammatory responses caused by oxidative stress often emerged upon exposure to GFNs
https://www.hindawi.com/journals/bmri/2021/5518999/
https://www.nature.com/articles/am20137
The treatment that should follow because of the toxicity of the substances used - (nanotechnology is used in masks, flu "vaccines", PCR "tests", chemtrails, foods, different drugs and of course and most importantly in C-19 injections), are antioxidants. There should be prevention and treatment of oxidative stress. Oxidative stress is the reason why people have low saturation, and as a result of severe oxidative stress, cells are destroyed, apoptosis occurs, that is, cell death and a further cascade of events:
clotting, organ failure, heart attacks, strokes, etc.
The virus (or just a regular flu that triggers these symptoms) seems to be pretty secondary to graphene toxicity. This is nanotechnology that crosses all blood barriers, is toxic, causes oxidative stress and injures/kills people.
This is also demonstrated here:
https://expose-news.com/2022/06/27/deadly-virus-bioweapon-or-damp-squib/
So, according to these studies on graphene toxicity, Covid is not a virus, but yes, people, poisoned by nanotechnology, because of free and oxy radicals present, are susceptible to infections or flu, and these infections, as additional oxidative stress - and strain on their already struggling organisms - can be fatal to them.
When Pfizer was asked how/where and for how long "Spike Protein" is produced, it admitted they had no idea. Spike Protein seems to be just a cover for injecting people and creating a fear of pandemics. This doesn't mean that a protein or even Spike Protein can't also be used in these vaccines, but if it is used, its functions seem to have nothing to do with creating antibodies to the "virus". But it may cause formation of misfolded proteins, AMYLOIDOSIS.
Thus, Covid is a misdiagnosed disease. It seems to be first and foremost graphene toxicity poisoning with all that come from this toxicity such as physical destruction, oxidative stress, DNA damage, inflammatory response, apoptosis, autophagy, and necrosis. Those are causing blood clots, organ failure, strokes, and many other injuries, including death. Graphene can result in acute inflammation response and chronic injury by interfering with the normal physiological functions of important organs. Studies regarding risks of graphene in the brain show that graphene application leads to harmful effects on brain tissue development and the atypical ultrastructure was observed in the brain. Graphene demonstrates its toxicity in the central nervous system and toxicity in reproduction and development system. In the animal studies the pregnant mice had abortions at all doses, and most pregnant mice died when the high dose was injected during late gestation and the development of offspring was delayed during the lactation period. The high dose reduces milk production and postpones the growth of offspring. The developmental toxicity of graphene induces structural abnormalities, growth retardation, behavioral and functional abnormalities, and even death. Graphene induces the lung injury with inflammatory cell infiltration, pulmonary edema and granuloma formation in the lungs. Graphene causes cytotoxic effects and mitochondrial injury, leads to inflammations, induces DNA damage, decreases cell adhesion and induces cell apoptosis - cell death. Graphene inserts between the base pairs of double-stranded DNA and disturb the flow of genetic information at the molecular level, which is the main cause of its mutagenic effect. It is hemotoxic, cytotoxic, cardiotoxic, neurotoxic, harmful to reproductive system. Graphene sharpened edges cause physical destruction...
There are other toxic ingredients and heavy metals in those vials:
https://www.aerzte-fuer-aufklaerung.de/wp-content/uploads/2022/07/Sammlung_erster_Ergebnisse_der_AG_Impfstoffe_Aufklärung_20220706.pdf
There is also toxic PEG. All of them will result in oxidative stress or acute oxidative stress.
https://pubmed.ncbi.nlm.nih.gov/30641367/ How graphene affects the misfolding of human prion protein: A combined experimental and molecular dynamics simulation study "Our simulations suggest that prion protein can be quickly and tightly adsorbed onto graphene together with the weak conformational rearrangement and may reorient when approaching the surface. The Van der Waals' force drive the adsorption process. In the induction of graphene, H1 and S2-H2 loop regions of prion become unstable and prion begins to misfold partially. Our work shows that graphene can induce the misfolding of prion protein and may cause the potential risk to biosystems."
So, for treatment - Glutathione precursors seem to be the most important because Glutathione is the master of all other antioxidants.
NAC helps dissolve amyloid plaque formations and blood clots and is one of the Glutathione precursors.
To address both oxidative stress and amyloid plaque formation, this is what would be helpful: Antioxidants!!! NAC, Zinc - and its ionophores, such, as Ivermectin, if available, Quercetin, Vitamin C-and others: Q10, K2MK7, Vitamin E, Resveratrol, Vitamin D, Beta Carotene, Nigella Sativa, Turmeric, etc.
https://www.hindawi.com/journals/neuroscience/2019/7547382/ - https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6500609/pdf/NEUROSCIENCE2019-7547382.pdf
N-acetylcysteine (NAC) may prevent strokes in people with hereditary cystatin C amyloid angiopathy (HCCAA), a rare genetic disorder
People with HCCAA have an average life expectancy of just 30 years, and most die within five years of their first stroke
NAC appears to work by preventing the formation of amyloid-producing proteins, which promote amyloid deposits linked to strokes
https://www.chop.edu/news/chop-researchers-find-supplement-prevents-strokes-patients-rare-genetic-disorder
The study by CHOP researchers suggests NAC may block the precipitation of amyloid plaque deposits, as well as help break up their formation
“Amyloids cannot precipitate without aggregating, so if we can prevent that aggregation with a drug [NAC] that is already available, then we could make an incredible difference in the lives of these patients.
Additionally, since we already have genetic testing available to identify these patients, we could conceivably give this treatment early in life and potentially prevent that first stroke from ever occurring.”
In the Pfizer Document - Postmarketing Experience, different Amyloidosis as an adverse result of vaccination is mentioned as well - including Cardiac Amyloidosis and Renal, Skin Amyloidosis - etc.
Here is the information what markers can be done to find out if oxidative stress is present: A limitation of our study, apart from the small sample size however, is that we were unable to do laboratory testing in our patients, including checking oxidative stress markers (lipid peroxides) as well as inflammatory markers (CRP, ferritin, D-dimer) and LDH which might demonstrate a change post GSH administration
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7172740/ - Efficacy of glutathione therapy in relieving dyspnea associated with COVID-19 pneumonia: A report of 2 cases
This is also summarized in my other post:
https://outraged.substack.com/p/short-summary
https://outraged.substack.com/p/this-is-not-spike-protein
Perhaps.....
https://forbiddenknowledgetv.net/sasha-latypova-with-katherine-watt-sars-cov-2-covid-19-is-a-us-dod-military-prototype-project/
CAN YOU IMAGINE IT BEING USED IN MASKS, TESTS, INJECTIONS, FOOD, AIR, ETC.?
Dr. Noack was a real expert on the subject.... As far as I remember, he looked at the spectroscopy results provided by Dr. Campra.
If Dr. Campra was right, Dr. Noack knew the exact answer.
https://www.nature.com/articles/srep07419
But even if it wasn't hydroxide, it's still extremely sharp and toxic. These people are guilty of mass murder.
However, Dr. Noack said it is not biodegradable. But there are studies that it is biodegradable.
Physical damage is one mechanism of damage caused by graphene.
"Furthermore, the sharpened edges of GNS may act as ‘blades’, inserting and cutting through bacterial cell membranes"
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5088662/#CR173
https://pubmed.ncbi.nlm.nih.gov/20925398/
Moreover, the extremely sharp edges of graphene nanowalls were found to damage the membrane of the microorganism by direct contact
https://www.researchgate.net/profile/Xiaolong-Tu/publication/266151786_Assessing_in_vivo_toxicity_of_graphene_materials_Current_methods_and_future_outlook/links/54ed849f0cf28f3e65358d15/Assessing-in-vivo-toxicity-of-graphene-materials-Current-methods-and-future-outlook.pdf
How does graphene damage viruses, bacteria and human cells? Graphene is a thin but
strong and conductive two-dimensional sheet of carbon atoms. There are three ways that it
can help prevent the spread of microbes: – Microscopic graphene particles have sharp
edges that mechanically damage viruses and cells as they pass by them.
http://hdreporter.com/health/9646-are-graphene-coated-face-masks-a-covid-19-miracle-or-another-health-risk
Toxicity of graphene-family nanoparticles: a general review of the origins and mechanisms -
PMC Published online 2016 Oct 31. doi: 10.1186/s12989-016-0168-y
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5088662/
In addition, several typical mechanisms underlying GFN toxicity have been revealed, for
instance, physical destruction, oxidative stress, DNA damage, inflammatory response,
apoptosis, autophagy, and necrosis. In these mechanisms, (toll-like receptors-) TLR-,
transforming growth factor β- (TGF-β-) and tumor necrosis factor-alpha (TNF-α) dependent pathways are involved in the signaling pathway network, and oxidative stress plays a
crucial role in these pathways.
Another study suggested that the irregular protrusions and sharp edges of the nanosheets could damage the plasma membrane, thus letting G entering the cell by piercing the phospholipid-bilayer (Li Y. et al., 2013). These features raise additional safety concerns, as free GRMs in the cytoplasm may lead to disruption of the cytoskeleton, impaired cell motility and blockade of the cell-cycle, similar to carbon nanotube-induced cytotoxicity. Exposed larvae displayed GO in the CNS and, most importantly, an induction of Parkinson’s disease-like symptoms such as disturbance of locomotor activity, dopaminergic neurons loss and formation of Lewy bodies. These effects were likely a consequence of mitochondrial damage and apoptosis through the caspase 8 pathway, in the presence of a more general metabolic disturbance
https://www.frontiersin.org/articles/10.3389/fnsys.2018.00012/full
Two recent studies give us a less than rosy angle. In the first, a team of biologists, engineers and material scientists at Brown University examined graphene’s potential toxicity in human cells. They found that the jagged edges of graphene nanoparticles, super sharp and super strong, easily pierced through cell membranes in human lung, skin and immune cells, suggesting the potential to do serious damage in humans and other animals.
https://newatlas.com/graphene-bad-for-environment-toxic-for-humans/31851/
Generally, GFNs may exert different degrees of toxicity in animals or cell models by following with different administration routes and penetrating through physiological barriers, subsequently being distributed in tissues or located in cells, eventually being excreted out of the bodies. This review collects studies on the toxic effects of GFNs in several organs and cell models. We also point out that various factors determine the toxicity of GFNs including the lateral size, surface structure, functionalization, charge, impurities, aggregations, and corona effect ect. In addition, several typical mechanisms underlying GFN toxicity have been revealed, for instance, physical destruction, oxidative stress, DNA damage, inflammatory response, apoptosis, autophagy, and necrosis.
https://www.sciencedaily.com/releases/2018/08/180823113613.htm
A human enzyme can biodegrade graphene
https://onlinelibrary.wiley.com/doi/10.1002/anie.201806906
Degradation of Single-Layer and Few-Layer Graphene by Neutrophil Myeloperoxidase
If you consider the implications of the DOD developing the Pfizer jab as a prototype countermeasure as described by Sasha Latypova, the DOD can instruct Pfizer to vary the contents of the inoculations as investigation of prototypes. The implication is that some vials will contain graphene and some not. Dr. Ryan Cole in conjunction with a mass spec lab recent analyzed a number of vials and found no graphene in any of them and found no mRNA is some of them as well (essential vials of saline). Others contained degraded mRNA with varying percentages of intact "jab spike protein" mRNA.. My interpretation is the jabs are a combination of different DOD prototypes and poor manufacturing quality control On another note, I appreciate all your links to research on graphene. Graphene is clearly toxic and it reportedly used in the limpid nanoparticles that come from a Chinese manufacturer (they state this on their website) as noted by Karen Kingston. With regard to ROS, toxins and viruses and pathogenic bacteria also elevate ROS. That graphene could be synergistic with pathogens in elevating ROS and misfolding of proteins and creating and inducing amyloid proteins is not beyond a possibility. Dr. Arne Bukhardt's autopsy biopsy slides clearly show amyloid infiltration of critical organs including the heart and brain and well as the spike protein "mRNA infections" triggering killer lymphocyte attacks on infected cells and consequent scarring.
Absolutely, I see two major factors contributing to injury and death:
amyloid plaque and oxidative stress.
https://outraged.substack.com/p/short-summary
https://outraged.substack.com/p/treatment-in-practice
https://outraged.substack.com/p/causes-of-injuries-and-deaths-from - With more information on amyloids:
The mere presence of toxic graphene, and similar components, can cause amyloidosis.
Amyloidosis is also a common complication after Covid-19 injections, as also confirmed by Pfizer documents listing various types of amyloidosis as possible post-vaccination complications in a Postmarketing Experience document: https://phmpt.org/wp-content/uploads/2021/11/5.3.6-postmarketing-experience.pdf
Amyloid arthropathy;
Amyloidosis;
Amyloidosis senile;
Cardiac amyloidosis;
Cerebral amyloid angiopathy;
Cutaneous amyloidosis;
Dialysis amyloidosis;
Gastrointestinal amyloidosis;
Hepatic amyloidosis;
Primary amyloidosis;
Pulmonary amyloidosis;
Renal amyloidosis;
Secondary amyloidosis;
Tongue amyloidosis; (“Covid tongue”)
So, the presence of toxic graphene can cause amyloidosis. However, if the technology used in these injections is also based on peptides (as has been widely reported in the scientific literature: Self-assembling peptide semiconductors | https://www.science.org/doi/10.1126/science.aam9756), the production of amyloid in the bone marrow can lead to severe amyloidosis and related complications and death, including the currently manifested so-called sudden death syndrome. The key question, therefore, is also what technology was used in these injections.
“Short peptides, specifically those containing aromatic amino acids, can self-assemble into a wide variety of supramolecular structures that are kinetically or thermodynamically stable; the representative models are diphenylalanine and phenylalanine-tryptophan. Different assembly strategies can be used to generate specific functional organizations and nanostructural arrays, resulting in finely tunable morphologies with controllable semiconducting characteristics. Such strategies include molecular modification, microfluidics, coassembly, physical or chemical vapor deposition, and introduction of an external electromagnetic field (EMF).
Self-assembling peptide nanomaterials may serve as an alternative source for the semiconductor industry because they are eco-friendly, morphologically and functionally flexible, and easy to prepare, modify, and dope. Moreover, the diverse bottom-up methodologies of peptide self-assembly facilitate easy and cost-effective device fabrication, with the ability to integrate external functional moieties. For example, the coassembly of peptides and electron donors or acceptors can be used to construct n-p junctions, and vapor deposition technology can be applied to manufacture custom-designed electronics and chips on various substrates.
The inherent bioinspired nature of self-assembling peptide nanostructures allows them to bridge the gap between the semiconductor world and biological systems, thus making them useful for applications in fundamental biology and health care research. Short peptide self-assemblies may shed light on the roles of protein semiconductivity in physiology and pathology. For example, research into the relationship between the semiconductive properties of misfolded polypeptides characteristic of various neurodegenerative diseases and the resulting symptoms may offer opportunities to investigate the mechanisms controlling such ailments and to develop therapeutic solutions. Finally, self-assembling short peptide semiconductors could be used to develop autonomous biomachines operating within biological systems. This would allow, for example, direct, label-free, real-time monitoring of a variety of metabolic activities, and even interference with biological systems.
Amyloid self-assembling peptides: Potential applications in nanovaccine engineering and biosensing https://onlinelibrary.wiley.com/doi/10.1002/pep2.24095
Peptide Semiconductor Times Are Coming | Nature Portfolio Bioengineering Community https://bioengineeringcommunity.nature.com/posts/37570-peptide-semiconductor-times-are-coming
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5457452/ “Peptide based nano-assemblies with their self-organizing ability has shown lot of promise due to their high degree of thermal and chemical stability, for biomaterial fabrication. Developing an effective way to control the organization of these structures is important for fabricating application-oriented materials at the molecular level. The present study reports the impact of electric and magnetic field-mediated perturbation of the self-assembly phenomenon, upon the chemical and structural properties of diphenylalanine assembly. Our studies show that, electric field effectively arrests aggregation and self-assembly formation, while the molecule is allowed to anneal in the presence of applied electric fields of varying magnitudes, both AC and DC. The electric field exposure also modulated the morphology of the self-assembled structures without affecting the overall chemical constitution of the material. Our results on the modulatory effect of the electric field are in good agreement with theoretical studies based on molecular dynamics reported earlier on amyloid forming molecular systems.”
“In last decade, there has been an increased focus on organic and bio-organic nano-assemblies. Peptide nanotubes, their physical properties, and assembly morphologies are extensively studied due to their excellent biocompatibility as well as functional and structural diversity. Many ordered supramolecular structures have been constructed using peptides as the building blocks. The most extensively utilized peptide-based building block is diphenylalanine (Phe-Phe or FF), which is the shortest bio-molecule known to self-assemble into ordered nanostructures. FF incidentally is also the core recognition motif of the β-amyloid polypeptide, a peptide associated with Alzheimer’s disease1. It can self-assemble into a variety of structures like microtubes, nanotubes2, microcrystals, nanofibers3, nanorods4, 5 and nanowires6.”
“The potential of these supramolecular structures have been utilized in diverse fields including nanofabrication, drug delivery vehicles7, bio-sensing8, energy storage devices, and hydrogels for tissue engineering”.
“One of the key challenges in the field of supramolecular chemistry has been controlling the self-assembly of molecules into ordered functional units. Previously, a number of strategies including pH mediated control21, 22, solvent mediated control23, covalent modifications24, vapour deposition25, 26, temperature27, surface28, 29, relative humidity30, symmetry31 and magnetite coating on the surface of nanotubes32 have been employed to regulate the architecture of diphenylalanine self-assemblies.”
Andrij Baumketner in a recent study, explored the feasibility of using external electric field to disaggregate amyloid fibrils, by inducing folding into an α-helical state reducing their β sheet conformation38. This is especially important because FF is the core recognition motif of β-amyloid segment. Here in this study, we attempt to confirm the effect of AC (Alternating Current) and DC (Direct Current) electric field on diphenylalanine self-assembly using experimental approach.
Different assembly strategies can be used to generate specific functional organizations and nano structural arrays, resulting in finely tunable morphologies with controllable semiconducting characteristics. Such strategies include molecular modification, microfluidics, subassembly, physical or chemical vapor deposition, and introduction of an external electromagnetic field.
When it comes to Dr. Cole, I trust Dr. Mihalcea:
https://anamihalceamdphd.substack.com/p/you-cant-find-what-you-are-not-looking?utm_source=post-email-title&publication_id=956088&post_id=90239968&isFreemail=true&utm_medium=email