My insulin requirements skyrocketed after being vaccinated. Here's why.
I'm a Type 2 diabetic and I use insulin to control my blood sugars. After being vaccinated, my insulin needs tripled. I was wondering what caused it. Now I know.
I have Type 2 diabetes which I control via insulin injections. Since being vaccinated, I’ve had to shoot three times as much insulin to keep my blood sugars under control.
This paper, Comprehensive investigations revealed consistent pathophysiological alterations after vaccination with COVID-19 vaccines explains it.
From the abstract, “Here, we report, besides generation of neutralizing antibodies, consistent alterations in hemoglobin A1c, serum sodium and potassium levels, coagulation profiles, and renal functions in healthy volunteers after vaccination with an inactivated SARS-CoV-2 vaccine… Altogether, our study recommends additional caution when vaccinating people with pre-existing clinical conditions, including diabetes, electrolyte imbalances, renal dysfunction, and coagulation disorders.”
This research was done with the Chinese Sinovac/Vero Cell vaccine, which uses inactivated virus.
I’d expect the results to be much worse for the US vaccines because I can’t imagine a mechanism of action that would make our vaccines better for this I’m not aware of any parameter where the mRNA vaccines are safer than an inactivated virus vaccine; the inactivated vaccines always give you a known dose of antigen whereas the mRNA vaccines give you a known dose of mRNA which creates an unknown amount of antigen.
They used 11 healthy volunteers with excellent HbA1c numbers. Look what happened to them:
They wrote:
To our surprise, quite consistent increases in HbA1c levels were observed in healthy volunteers, regardless of whether they belonged to cohort A or B. By day 28 post the 1st inoculation, three out of 11 individuals reached the prediabetic range (Fig. (Fig.2c).2c). By days 42 and 90, medium HbA1c levels appeared to revert back, yet were still significantly higher than those before vaccination.
Now, imagine what happens to someone with high HbA1c. Well, we have to imagine it because the FDA never required any of the drug companies to measured these parameters before and after. They didn’t want to look at these before they approved these safe and effective vaccines!
A big thanks to Dr. Sabine Hazan for bringing it to my attention. She’s awesome.
Too bad they don’t warn you about this, isn’t it?
Their response could be, “nothing to see here folks, just move along…” as suggested here:
But no, it’s much more sinister than that… Check this out:
You simply cannot make this up folks. You can see that ad right on the CDC website.
Has this happened to anyone else? Please let me know in the comments.
Update 11/26/21
Someone on Mathew Crawford's channel asked to pass this onto you. SUMMARY: "covid spike causes bile leakage into the blood. The bile leakage then injures the pancreas. Still speculative, but should be easy to test bile salts before/after vaccination, and pancreatic function...."
Detail:
I’ve seen other stories of diabetic complications as well. I thought it was due to ROS/leakage in the pancreas itself, but it looks like it is actually due to bile acids leaking from liver bile ducts via ACE2 in the ducts.
So covid spike causes bile leakage into the blood. The bile leakage then injures the pancreas.
— papers --
Recapitulation of SARS-CoV-2 infection and cholangiocyte damage with human liver ductal organoids
Specific ACE2 Expression in Cholangiocytes May Cause Liver Damage After 2019-nCoV Infection
Human Insulin Resistance Is Associated With Increased Plasma Levels of 12α-Hydroxylated Bile Acids
Insulin-secreting beta cells do not have high levels of ACE2, so are probably not affected directly by spike protein. But they have a rich blood supply, and the micro-vasculature has lots of ACE2. https://pubmed.ncbi.nlm.nih.gov/33207245/
Beta cells are highly sensitive to reactive oxygen species (ROS), because they have to sample blood for glucose levels and that sampling itself creates ROS. So spike protein triggers ROS in the microvasculature, killing some of the beta cells.
When beta cells die, it's a double whammy. Pancreas islets are a mix of beta (insulin) and alpha (glucagon) cells. Beta cells sample for glucose, and if they detect it they turn off glucagon secretion. When the beta cells die, there's no shut-off for the alpha cells, and baseline glucagon levels increase, which tells your liver to convert stored glycogen to glucose. This constant baseline increase in glucagon requires even more insulin to be produced by the remaining beta cells (because insulin receptors respond to the net insulin:glucagon balance), so they run even higher ROS and accelerated death rates.
Beta cells do regrow, albeit slowly, if you can reduce ROS enough. I used to be 50 pounds overweight and borderline diabetic, now have normal weight and no blood sugar issues afaik, but it took years to figure out how to resolve it. Partly learning enough and sorting through all the conflicting information, partly time to heal/regrow.
Type II diabetes does not simply affect overweight people or people who “eat too many carbs.” There is a hybrid diabetes that is classified as Type II, but these folks are often of completely normal weight and yet, need insulin, not just oral hypoglycemic agents. Don’t rag on Steve or his diabetes “causes.” He shared something intimate with all of us fand the body is a complex machine as are its genetics. Let’s be supportive of each other. Every “body” is different and responds in complexity with one’s environment and genetics, and habits. Best of health to you, Steve. We are truly grateful for your tireless work for us all. You are a true humanitarian.