I was just alerted to this interview. The inattention being paid this is not good. Cole didn't say but infers that these clots are fibrin that agglomerates in the presence of S2 Spike. You can get S2 Spike from either infection or vaccine. More with vaccine and more with each dose. But still. If even natural infection does this tha…
I was just alerted to this interview. The inattention being paid this is not good. Cole didn't say but infers that these clots are fibrin that agglomerates in the presence of S2 Spike. You can get S2 Spike from either infection or vaccine. More with vaccine and more with each dose. But still. If even natural infection does this that means EVERYONE is blood clotted to some degree or another. Are they looking for a compound that dissolves these? If not why not. What's going on here.
Kell, D. B., Laubscher, G. J., & Pretorius, E. (2022). A central role for amyloid fibrin microclots in long COVID/PASC: origins and therapeutic implications. Biochemical Journal, 479(4), 537-559.
I was just alerted to this interview. The inattention being paid this is not good. Cole didn't say but infers that these clots are fibrin that agglomerates in the presence of S2 Spike. You can get S2 Spike from either infection or vaccine. More with vaccine and more with each dose. But still. If even natural infection does this that means EVERYONE is blood clotted to some degree or another. Are they looking for a compound that dissolves these? If not why not. What's going on here.
It's S1.
I think you'll fine this paper useful.
https://portlandpress.com/biochemj/article/479/4/537/230829
Kell, D. B., Laubscher, G. J., & Pretorius, E. (2022). A central role for amyloid fibrin microclots in long COVID/PASC: origins and therapeutic implications. Biochemical Journal, 479(4), 537-559.