You can post all you want but most people on this forum will just laugh at you. You are wasting your time. And as a clinical scientist and someone with a doctorate in microbiology everything you said about coronaviruses is fiction. But you do feel compelled to attach web links to sources that receive their funding from the vaccine manufacturers?
You can post all you want but most people on this forum will just laugh at you. You are wasting your time. And as a clinical scientist and someone with a doctorate in microbiology everything you said about coronaviruses is fiction. But you do feel compelled to attach web links to sources that receive their funding from the vaccine manufacturers?
Ok, doctor. Tell me what your thoughts are on this.
“We specifically highlight the role of the four above-mentioned cytokines (IL-6, IL-10, IL-18 and IL-27), as they seem to be ‘constant’ markers for COVID-19 infection. Their concentrations rise independently of the genetic variant of the virus, potentially marking the dysregulation of immune responses to SARS-CoV-2 infection. This study is a successor for our previous work focused on the chemokine profiling of different SARS-CoV-2 genetic variants; overall, it is becoming more evident that the evolution of the virus brings new challenges to immunology.” (9)
A case study of 58 year old man experiencing a rapid increase in cytokins after vaccination, with BNT162b2 (tozinameran), an mRNA COVID-19 vaccine, had a persistent elevation of IL-2R, IL-2, IL-16 and IL-18 on day 12 of admission indicating sustained T cell activation.(10)
Hypothesis:
COVID can cause a significant increase of inflammatory cytokines, and potentially to a less significant degree, the mRNA vaccine booster, particularly if taken recently after an active (acute) infection, during a persistent (chronic) infection, or predisposed to any of the more than 80 types of autoimmune diseases that affect a wide range of body parts. There may be an increased risk of an autoimmune response for a few days to a weeks from vector and mRNA based vaccinations. The risk from COVID is many times greater but it appears there is the potential for this to occur with vector and mRNA based vaccines in a subset of people.
The "potential" adverse risk associated with taking a vaccine booster dose too soon after the last dose of mRNA vaccine, "Of note, cytokine production in CD8+ T cells was boosted in donors that received three doses of mRNA vaccine."(12)
“Cytokine-positive CD8+ memory T cells (CD45RO+IFN-γ+CD8+ T cell) were observed (21% for mRNA vaccinated, 43% for vector vaccinated, 0% for heterologous vaccinated)”(12) In the spring of 2021, the Johnson and Johnson vaccine, Janssen was paused over concerns over rare blood clots. A study comparing adverse reactions to Sinopharm and Pfizer–BioNTech COVID-19 vaccines said, “Participants who received the Pfizer–BioNTech vaccine had significantly higher frequencies of all types of adverse reactions”(13)
Similar to COVID, these vaccines could “theoretically” cause vascular dysfunction in some people. “Accumulating evidence suggest that vascular inflammation and inflammatory cytokines play a role in the vascular dysfunction associated with vascular disease.”(11) In theory, the sudden burst of these inflammatory cytokines in someone with pre-existing conditions or an undiagnosed persistent infection , it may cause vascular dysfunction for a few days or weeks.
You can post all you want but most people on this forum will just laugh at you. You are wasting your time. And as a clinical scientist and someone with a doctorate in microbiology everything you said about coronaviruses is fiction. But you do feel compelled to attach web links to sources that receive their funding from the vaccine manufacturers?
Ok, doctor. Tell me what your thoughts are on this.
“We specifically highlight the role of the four above-mentioned cytokines (IL-6, IL-10, IL-18 and IL-27), as they seem to be ‘constant’ markers for COVID-19 infection. Their concentrations rise independently of the genetic variant of the virus, potentially marking the dysregulation of immune responses to SARS-CoV-2 infection. This study is a successor for our previous work focused on the chemokine profiling of different SARS-CoV-2 genetic variants; overall, it is becoming more evident that the evolution of the virus brings new challenges to immunology.” (9)
A case study of 58 year old man experiencing a rapid increase in cytokins after vaccination, with BNT162b2 (tozinameran), an mRNA COVID-19 vaccine, had a persistent elevation of IL-2R, IL-2, IL-16 and IL-18 on day 12 of admission indicating sustained T cell activation.(10)
Hypothesis:
COVID can cause a significant increase of inflammatory cytokines, and potentially to a less significant degree, the mRNA vaccine booster, particularly if taken recently after an active (acute) infection, during a persistent (chronic) infection, or predisposed to any of the more than 80 types of autoimmune diseases that affect a wide range of body parts. There may be an increased risk of an autoimmune response for a few days to a weeks from vector and mRNA based vaccinations. The risk from COVID is many times greater but it appears there is the potential for this to occur with vector and mRNA based vaccines in a subset of people.
The "potential" adverse risk associated with taking a vaccine booster dose too soon after the last dose of mRNA vaccine, "Of note, cytokine production in CD8+ T cells was boosted in donors that received three doses of mRNA vaccine."(12)
“Cytokine-positive CD8+ memory T cells (CD45RO+IFN-γ+CD8+ T cell) were observed (21% for mRNA vaccinated, 43% for vector vaccinated, 0% for heterologous vaccinated)”(12) In the spring of 2021, the Johnson and Johnson vaccine, Janssen was paused over concerns over rare blood clots. A study comparing adverse reactions to Sinopharm and Pfizer–BioNTech COVID-19 vaccines said, “Participants who received the Pfizer–BioNTech vaccine had significantly higher frequencies of all types of adverse reactions”(13)
Similar to COVID, these vaccines could “theoretically” cause vascular dysfunction in some people. “Accumulating evidence suggest that vascular inflammation and inflammatory cytokines play a role in the vascular dysfunction associated with vascular disease.”(11) In theory, the sudden burst of these inflammatory cytokines in someone with pre-existing conditions or an undiagnosed persistent infection , it may cause vascular dysfunction for a few days or weeks.
What are your thoughts on that?