Even if you’re correct that the virus is causing all these problems more than the vaccines are (the evidence suggests otherwise), you still have a huge problem. The jabs don’t freaking work! Australia, New Zealand, and Scandinavia are having never ending waves of covid despite (or probably because of) high vaccination rates. So all these jabbed people are at risk of long covid anyway!
Even if you’re correct that the virus is causing all these problems more than the vaccines are (the evidence suggests otherwise), you still have a huge problem. The jabs don’t freaking work! Australia, New Zealand, and Scandinavia are having never ending waves of covid despite (or probably because of) high vaccination rates. So all these jabbed people are at risk of long covid anyway!
The vaccine reduced the odds of Long COVID by about 15% for a few months. The vaccines only helped prevent severe disease and death by inducing a CD8 T-cells. The circulating T-cells wane within 6 to 10 months. Depending on T-cells means that the virus has time to circulate and infiltrate before infecting cells, at which point the T-cells respond to take out the infected cells. That means that it has time to infect organs and other areas out of reach from the immune response., enabling it to persist. The XXB variants are the most antibody evasive variants to date, and can infect most cells of the immune system. Since it can so effectively suppress the immune response, people are having less symptoms. Most symptoms are a result of the fight. No fight, less symptoms. That's is how insidious this has become.
Persistent infections cause an ongoing T-cell response and inflammatory response. That leads to vascular issues and autoimmune issues.
Coronaviruses have a history of causing vascular dysfunction and blood clotting that could lead to heart attacks and strokes.
“Coagulation disorders in coronavirus infected patients: COVID-19, SARS-CoV-1, MERS-CoV and lessons from the past” (2)
They concluded, “The dysregulation of the coagulation cascade and the subsequent formation of intra-alveolar or systemic fibrin clots are prominent findings in coronavirus infections associated with severe respiratory disease, and have been demonstrated in both humans and animal models. They can be attributed to the prothrombotic response, which attempts to prevent diffuse alveolar hemorrhage, but can instead result in overt clot formation with detrimental effects in patient recovery and survival.”
COVID has a history of causing vascular dysfunction, blood clotting and strokes prior to vaccines.
Studies from 2020 demonstrate how COVID affects the vascular system before anyone took the vaccines.
There are a lot of studies that get into much more detail between 2020 and today but this is to demonstrate that we already knew about this occurring before people started taking vaccines. Together, with all the evidence below, we show that COVID is the primary driver of the excess deaths. This is largely due to patients that die from a stroke or a heart attack weeks or months after the initial infection. As a result, COVID likely went undetected at the time of admission and thus was not recorded.
An analysis of over 17,000 cardiovascular events after COVID-19, using the UK Biobank looked at cases between March 2020 and 2021. It showed that the top 3 cardiovascular events were VTE, followed by heart failure and stroke. They noted the risks were greatest in the first 30 days after infection but that risks remained higher afterward. (3)
August 2020, Platelet and Vascular Biomarkers Associate With Thrombosis and Death in Coronavirus Disease “Our findings are consistent with recent reports of platelet hyperactivity in patients with COVID-19. We extend those finding and demonstrate that biomarkers of platelet activation are associated with thrombosis or death in patients hospitalized with COVID-19.”(4)
In a study published, on August 16, 2020, they noted that "coronaviruses are known to affect the cardiovascular system.” This was an early warning when they noted that COVID-19 itself might aggravate the myocardial injury, by causing the “release of multiple cytokines and chemokines that can not only lead to vascular inflammation and plaque instability but also to myocardial inflammation.”(5)
Additional References from 2020:
Coagulation abnormalities and thrombosis in patients with COVID-19.
Lancet Haematol.2020;7:e438-e440 View in Article, Scopus (874),
Thrombocytopenia is associated with severe coronavirus disease 2019 (COVID-19) infections: a meta-analysis. Clin Chim Acta.2020; 506:145-148
Immune Evasion and Suppression
We already have studies showing how COVID can infect CD-4 T-cells, dendritic cells, and neutrophils, so the question of whether or not COVID can weaken the immune system by depleting these cells has been answered.
We know that COVID has been listed as one of the leading causes of Lymphocytopenia. "People with lymphocytopenia experience recurrent infections or develop infections with unusual organisms & it is a risk factor for the development of cancers & for autoimmune disorders." (7)
COVID impacts everyone’s immune system to varying degrees. Some people recover faster than others, but it can take weeks to more than eight months to recover. (8)
There has been a lot of understandable confusion. The reality is that COVID is the primary driver of the excess deaths. The heart attacks and strokes aren't being recorded as COVID related when in fact they are. That's what is behind the excess deaths.
In a data analysis from the Smidt Heart Institute at Cedars-Sinai, published on September 29, 2022, they ran the data and showed that increases in heart attack mortality have coincided with increases in COVID-19 infection, even during the pandemic's assumed milder Omicron phase. Also, the increase was most noticeable in people between the ages of 25 and 44, who are not usually thought to be at high risk for a heart attack. This age group is less vaccinated than the older adults, who tend to be at the highest risk for heart attacks.
They found that acute myocardial infarction deaths during the pandemic increased across all age groups but were most significant in the youngest group they looked at, which was ages 25 to 44. In 2021, the "observed" death rates from heart attacks were 29.9% higher than what was "predicted" for adults 25–44, 19.6% higher for adults 45–64, and 13.7% higher for adults 65 and older. The most vaccinated older adults are dying less often than younger adults due to acute myocardial infarction.
The 25–44 age group is more likely to be re-infected by COVID through their jobs and by their school-age children.
In the year before the pandemic, there were 143,787 heart attack deaths; within the first year of the pandemic, before vaccines, this number had increased by 14% to 164,096.
You can read much more detail with links to all the references.
Even if you’re correct that the virus is causing all these problems more than the vaccines are (the evidence suggests otherwise), you still have a huge problem. The jabs don’t freaking work! Australia, New Zealand, and Scandinavia are having never ending waves of covid despite (or probably because of) high vaccination rates. So all these jabbed people are at risk of long covid anyway!
The vaccine reduced the odds of Long COVID by about 15% for a few months. The vaccines only helped prevent severe disease and death by inducing a CD8 T-cells. The circulating T-cells wane within 6 to 10 months. Depending on T-cells means that the virus has time to circulate and infiltrate before infecting cells, at which point the T-cells respond to take out the infected cells. That means that it has time to infect organs and other areas out of reach from the immune response., enabling it to persist. The XXB variants are the most antibody evasive variants to date, and can infect most cells of the immune system. Since it can so effectively suppress the immune response, people are having less symptoms. Most symptoms are a result of the fight. No fight, less symptoms. That's is how insidious this has become.
Persistent infections cause an ongoing T-cell response and inflammatory response. That leads to vascular issues and autoimmune issues.
Coronaviruses have a history of causing vascular dysfunction and blood clotting that could lead to heart attacks and strokes.
“Coagulation disorders in coronavirus infected patients: COVID-19, SARS-CoV-1, MERS-CoV and lessons from the past” (2)
They concluded, “The dysregulation of the coagulation cascade and the subsequent formation of intra-alveolar or systemic fibrin clots are prominent findings in coronavirus infections associated with severe respiratory disease, and have been demonstrated in both humans and animal models. They can be attributed to the prothrombotic response, which attempts to prevent diffuse alveolar hemorrhage, but can instead result in overt clot formation with detrimental effects in patient recovery and survival.”
COVID has a history of causing vascular dysfunction, blood clotting and strokes prior to vaccines.
Studies from 2020 demonstrate how COVID affects the vascular system before anyone took the vaccines.
There are a lot of studies that get into much more detail between 2020 and today but this is to demonstrate that we already knew about this occurring before people started taking vaccines. Together, with all the evidence below, we show that COVID is the primary driver of the excess deaths. This is largely due to patients that die from a stroke or a heart attack weeks or months after the initial infection. As a result, COVID likely went undetected at the time of admission and thus was not recorded.
An analysis of over 17,000 cardiovascular events after COVID-19, using the UK Biobank looked at cases between March 2020 and 2021. It showed that the top 3 cardiovascular events were VTE, followed by heart failure and stroke. They noted the risks were greatest in the first 30 days after infection but that risks remained higher afterward. (3)
August 2020, Platelet and Vascular Biomarkers Associate With Thrombosis and Death in Coronavirus Disease “Our findings are consistent with recent reports of platelet hyperactivity in patients with COVID-19. We extend those finding and demonstrate that biomarkers of platelet activation are associated with thrombosis or death in patients hospitalized with COVID-19.”(4)
In a study published, on August 16, 2020, they noted that "coronaviruses are known to affect the cardiovascular system.” This was an early warning when they noted that COVID-19 itself might aggravate the myocardial injury, by causing the “release of multiple cytokines and chemokines that can not only lead to vascular inflammation and plaque instability but also to myocardial inflammation.”(5)
Additional References from 2020:
Coagulation abnormalities and thrombosis in patients with COVID-19.
Lancet Haematol.2020;7:e438-e440 View in Article, Scopus (874),
Thrombocytopenia is associated with severe coronavirus disease 2019 (COVID-19) infections: a meta-analysis. Clin Chim Acta.2020; 506:145-148
Immune Evasion and Suppression
We already have studies showing how COVID can infect CD-4 T-cells, dendritic cells, and neutrophils, so the question of whether or not COVID can weaken the immune system by depleting these cells has been answered.
We know that COVID has been listed as one of the leading causes of Lymphocytopenia. "People with lymphocytopenia experience recurrent infections or develop infections with unusual organisms & it is a risk factor for the development of cancers & for autoimmune disorders." (7)
COVID impacts everyone’s immune system to varying degrees. Some people recover faster than others, but it can take weeks to more than eight months to recover. (8)
There has been a lot of understandable confusion. The reality is that COVID is the primary driver of the excess deaths. The heart attacks and strokes aren't being recorded as COVID related when in fact they are. That's what is behind the excess deaths.
In a data analysis from the Smidt Heart Institute at Cedars-Sinai, published on September 29, 2022, they ran the data and showed that increases in heart attack mortality have coincided with increases in COVID-19 infection, even during the pandemic's assumed milder Omicron phase. Also, the increase was most noticeable in people between the ages of 25 and 44, who are not usually thought to be at high risk for a heart attack. This age group is less vaccinated than the older adults, who tend to be at the highest risk for heart attacks.
They found that acute myocardial infarction deaths during the pandemic increased across all age groups but were most significant in the youngest group they looked at, which was ages 25 to 44. In 2021, the "observed" death rates from heart attacks were 29.9% higher than what was "predicted" for adults 25–44, 19.6% higher for adults 45–64, and 13.7% higher for adults 65 and older. The most vaccinated older adults are dying less often than younger adults due to acute myocardial infarction.
The 25–44 age group is more likely to be re-infected by COVID through their jobs and by their school-age children.
In the year before the pandemic, there were 143,787 heart attack deaths; within the first year of the pandemic, before vaccines, this number had increased by 14% to 164,096.
You can read much more detail with links to all the references.
https://tactnowinfo.substack.com/p/what-is-causing-the-excess-deaths