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Stephen David Watson's avatar

As an M.D., Ph.D. anesthesiologist and former E.R. physician of 39 years, I have read significant misinterpretation of the events surrounding the collapse of Hamlin. While I do not purport to be a proverbial expert, I believe my assessments are valid. The possible diagnoses I discount include commotio cordis, intracranial/cardiac or intra-thoracic aneurysm that ruptured.

The initial diagnosis by a number of cardiologists and other physicians was that this was a case of commotio cordis. Recognize that the acute and sharp trauma to the chest wall and the underlying heart at a precise point in the cardiac cycle (20msec total interval during the T wave which represents ventricular repolarization) causes IMMEDIATE change from a sinus rhythm (normal) to ventricular fibrillation (v. fib.). When v. fib. occurs, no significant cardiac output occurs and within several seconds at the most, the brain and heart become hypoxic and cease to function normally. In other words, the onset of v. fib. results in a dead person that has only several minutes at the most IF given adequate CPR and typically ventilation to keep the dying brain alive and a sinus rhythm restored. If the sinus rhythm is not immediately restored, the brain rapidly loses the potential to be salvaged. No one can say exactly how few seconds occur in the conscious person who converts into v. fib. before they become unconscious. I seriously doubt more than 3-4 seconds at the most would pass before the individual would be aware that something was drastically wrong and visibly express alarm. Note that last year there was a 500% increase in the incidence of top-level, professional soccer players in the European leagues who dropped to the playing pitch, most of them in v. fib./dying. The coverage of the incident with Hamlin shows the hit (not apparently unusually violent) and virtually immediately stood upright, adjusted his helmet as if nothing was wrong and THEN collapsed. If this was c. c., he would have recognized immediately after the hit while still on the ground that something was drastically wrong and would likely have not stood up or would have exhibited alarm or confusion as he stood, if he still could have done so. How many seconds did it take for him to stand upright apparently in no distress, adjust his helmet and THEN collapse? Too many I propose to have occurred because of c. c.

The second consideration of a possible diagnosis was and is a cardiac aneurysm or intra-cranial/ brain aneurysm rupture. There is clear evidence for a significant incidence of development of aneurysms in the heart and also the brain after the Covid mRNA "vaccines." The problem with either of these possible diagnoses is that rupture of either would cause IMMEDIATE recognition of severe chest pain (cardiac event) or "the worst headache of their life" if it was a brain aneurysm that ruptured. The video evidence shows that the patient had a significant interval of seemingly normal cardiac and brain function through the act of standing up and adjusting his helmet BEFORE he collapsed. A more likely cause for his deterioration from a sinus rhythm to ventricular fibrillation is that the Covid "vaccinations" which cause a numerically greater load of spike protein to be developed and is deposited (shown in post-mortem studies) in many tissues of the body including the heart. This creates an inflammatory reaction involving the endothelium of blood vessels in different areas of the body. That irritation/inflammation of the endothelium leads to the potential for stimulation of the platelets to change their function in the same manner that they do when a bleeding vessel occurs, ie. they collect and block the hole in vessel to stop the leak. In this scenario, the blockage caused by the platelet aggregation literally blocks the coronary vessel in exactly the same manner as occurs with a typical heart attack. If the vessels are not "overwhelmed" and still functioning normally, a blood clot will not necessarily form. What can and apparently does occur is that the myocardial cells themselves develop abnormal activity or function and likely set-off the change to v. fib. and death. Recall the dramatic increased incidence of myocarditis (this latter scenario) over the last 1.75 years, particularly in young men. The moment a significant arteriole occlusion occurs, hypoxic heart tissue ensues and the cascade happens that can and likely leads to v. fib. and death. These two latter causes for Hamlin's collapse are likely the reason for his terrible event. I certainly hope and pray that this young man will recover. The prognosis will become more certain over the next couple of days. The reality is that, as Steve Kirsch states, the incidence of acute cardiac arteriole occlusion (heart attack as above) or myocarditis (as in the latter example) need to be clinically and post-mortem examined in depth to find out whether or not the mRNA "vaccines" are causative or not. There should not be any hesitation to do so because that has always been the hallmark of good medical practice. May God support and bless the family of Hamlin and give Hamlin the healing touch that he so desperately needs.

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Steve Kirsch's avatar

We had to disable comments for free subscribers due to abuse by one individual who keeps spamming my substack. If you are a paid subscriber, you can still comment.

I will be in touch with the CEO of Substack to look into fixing this issue.

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